Beta-N-methylamino-L-alanine (BMAA), a cyanobacterial neurotoxin accumulating inside marine trophic chains, has been deemed responsible for Aβ plaques and dystrophic neurites in bottlenose and common dolphins (Delphinus delphis) stranded along the Atlantic USA seaboard [6], with a TAR DNA-binding protein 43 (TDP-43) proteinopathy and AD-like brain lesions having been additionally reported in a BMAA-exposed harbor porpoise (Phocoena phocoena) [7]. Here, TARDBP is linked to Alzheimer disease.