The results obtained were a negative association between the oral presence of the bacterium and gastric infections/complications between the group of cases and controls; but on the contrary, it was shown that the pathogenic genes of oral Hp can increase the severity of gastric infection, increasing the risk of developing conditions such as gastritis, gastro-oesophageal reflux, and the mucosa of the upper gastrointestinal tract normal with lax esophageal sphincters [16]. The gene discussed is HP; the disease is gastritis.