HSPD1 and coronary artery disorder: Matsuura et al. proposed that molecular mimicry involving H. pylori heat shock protein 60 (Hp-HSP60) may contribute to the pathogenesis of CAD by stimulating Th1 lymphocytes to produce IL-12 and INF-γ, or by activating macrophages crucial in the formation of atherosclerotic plaques [53].