We used a TaqMan RT-PCR-based high-throughput platform to analyze a variety of genes involved in APP and Aβ-42 metabolism (Table S3) in liver samples from patients with an MAS of 1–4 (steatosis) or 5–8 (MASH), and from patients with histopathologically proven non-diseased livers (MAS = 0). The gene discussed is APP; the disease is steatosis.