Other aspects of NFκB function in cancer, such as the different interactions of NFκB with oncogenes, and the resulting effects on cell fate and cell phenotype have been exhaustively studied and reviewed elsewhere, [23,24,25,26,27,28,29,30,31,32,33] and will only be mentioned here in connection with the key distinguishing feature of relapsed cancer, namely the abnormal response to NFκB inducing cell stress. This evidence concerns the gene NFKB1 and cancer.