They exhibit transient IL-9 production, heightened IL-13, and IL-5 secretion [7], a tendency to express the skin-homing receptor known as cutaneous lymphocyte-associated antigen (CLA) [8,9], and are typically located in skin lesions of atopic dermatitis (AD), psoriatic, and contact dermatitis patients [7,9]. This evidence concerns the gene SELPLG and atopic eczema.