A separate bioinformatic analysis by Almotiri et al. demonstrated that both ZEB1 and ZEB2 in human AML are significantly higher in the leukemic blast population than in the bulk tumor population, suggesting that these transcription factors may be diluted in the bulk RNA-sample analysis as well as in overall levels of expression used in the Kaplan–Meier survival curves [130]. The gene discussed is ZEB2; the disease is neoplasm.