In the first attempt to distinguish compensated cirrhosis (precursor condition of decompensated cirrhosis with clinically evident complications like ascites, variceal hemorrhage, hepatic encephalopathy, and jaundice) from a heterogeneous group of CLD patients using VOCs, a set of 11 volatiles outperformed a combination of 5 serum markers (GGT, ALT, bilirubin, albumin, and thrombocytes). The gene discussed is GPT; the disease is congenital secretory chloride diarrhea 1.