The modulation of SNHG1 expression has far-reaching implications in the immune landscape of malignant tumors, namely, HCC, potentially serving as a double-edged sword that not only alters the infiltration and function of various immune cells but also influences the immune escape mechanisms of tumor cells, thereby offering a promising target for therapeutic interventions aimed at enhancing immunogenicity and inhibiting tumor progression. Here, SNHG1 is linked to neoplasm.