In fact, the absence of IL-10 has been shown to be associated with the prevention of intracellular inflammasome assembly and dendritic cell apoptosis, allowing for more efficient antigen presentation and consequently an intensified Th1 cell immune response, which is essential for counteracting chlamydial infections in IL-10 knockout mice [8,9]. Here, IL10 is linked to chlamydia trachomatis infectious disease.