It inhibits the overexpression of oncogenes and signaling pathways involved in PCa, such as Bcl-2 [175], androgen receptor (AR) signaling, EGFR, HER2, Cyclin D1, COX-2, MMPs, Akt, NF-κB, AP-1, STAT3, MAPK, JAK/STAT, and PI3K/Akt/mTOR. This evidence concerns the gene AKT1 and posterior cortical atrophy.