However, in the LHV, despite no alterations in magnetic parameters in all groups investigated vs. the control group, we found upregulated gene expressions of genes involved in iron metabolism: Tfr1 (the receptor for transferrin-bound iron entry into cells, its elevated expression serves as a tissue iron deficiency marker), Dmt1 (the major divalent iron transporter contributing to divalent iron uptake in most types of cells), and Hamp (the gene encoding hepcidin, a hormone that inhibits iron exporter ferroportin function) determined as the main effects of stress and DMF, respectively. The gene discussed is SLC11A2; the disease is nutritional disorder.