Another aspect reported in the literature was that SARS-CoV-2 infection causes a dysregulated cytokine response with elevated expression of IFN-γ and proinflammatory cytokines such as interleukin (IL-1, IL-6, IL-7, IL-10) and tumour necrosis factor-alpha, which in turn potentially be exacerbated by the shift from Th1 to Th2 response seen in SLE [55]. Here, IFNG is linked to systemic lupus erythematosus.