UPF3A and infection: They revealed that Arabidopsis UPF1 and UPF3 were destroyed via ubiquitination at an early stage of PstDC3000 infection, which led to the steady‐state levels and the stability of R transcripts in bacterial‐infected plants,[45] pointing to an additional layer of plasticity between the PRRs‐ and R‐induced immunity by NMD.