In cisplatin‐induced mouse AKI, stevioside inhibits oxidative stress, inflammation, and apoptosis by regulating the expression of cisplatin‐activated extracellular signal‐regulated kinases 1 and 2 and signal transducer and activator of transcription 3 (STAT3), thereby reducing the nephrotoxicity of cisplatin.27 Here, STAT3 is linked to acute kidney injury.