Researchers discovered that the global mRNA level of m6A rose considerably via the Epithelial mesenchymal transition process, and they pinpointed Snail as a METTL3‐mediated m6A alteration target, which cooperates with YTHDF1 to encourage Snail's protein translation and contribute to HCC metastasis.56 This evidence concerns the gene METTL3 and hepatocellular carcinoma.