According to Chen et al., METTL3 is frequently up‐regulated in human HCC and encourages colony formation, growth, and migration of HCC cells in culture as well as tumorigenicity, growth, and lung metastasis of HCC in vivo by suppressing SOCS2 mRNA, whose degradation depends on the m6A reader protein‐dependent pathway.55 The gene discussed is SOCS2; the disease is hepatocellular carcinoma.