According to Chen et al., METTL3 is frequently up‐regulated in human HCC and encourages colony formation, growth, and migration of HCC cells in culture as well as tumorigenicity, growth, and lung metastasis of HCC in vivo by suppressing SOCS2 mRNA, whose degradation depends on the m6A reader protein‐dependent pathway.55 This evidence concerns the gene METTL3 and hepatocellular carcinoma.