This included HRAS (11p15) amplification on chromothriptic chromosome 11, KRAS (12q12) and ERBB3 (12q13) amplification on chromosome 12, as well as deletions of key carcinoid-associated tumor suppressor genes MEN1 (11q13) and ARID1A (1p36) on chromosomes 11 and 1, respectively. The gene discussed is KRAS; the disease is carcinoid tumor.