FGF23 and periodontitis: Kajiwara et al. [6] described in their study that the presence of P. gingivalis lipopolysaccharide (Pg-LPS) is related to the accumulation of FGF23 in the renal tubules and glomeruli, implying that Pg-LPS enhanced FGF23 accumulation in the renal tubule and thus increase its concentration in the circulation. This indicates a potential pathway through which periodontitis could influence FGF23 levels.