Accumulating evidence has shown that m6A-related enzymes regulate liver fibrosis; for example, Chaiet al. [16] revealed that Mettl3 deficiency attenuated HSC activation and CCl4-induced liver fibrosis, and Liuet al. [17] reported that the m6A reader YTHDF3 alleviated CCl4-induced liver fibrosis. Here, YTHDF3 is linked to Hepatic fibrosis.