Notably, unlike the phenotype caused by SLC39A9 knockout, we found that C1GalT1 knockout in Huh7.5.1-VP30 cells did not impair EBOVΔVP30-EGFP infection (Fig 2A), indicating that SLC39A9-mediated glycosylation has limited effect on EBOV infection. The gene discussed is C1GALT1; the disease is infection.