The concentration of TG is elevated during the postprandial phase (40) though the increment is modest in majority of patients, between 0.14–0.3 mmol/L (12–27 mg/dl) (41) Non-fasting rather than fasting TG concentration is independently associated with atherosclerosis and incident future ASCVD events independent of other ASCVD risk factors, lipid parameters and insulin resistance (45, 47, 48). This evidence concerns the gene INS and atherosclerosis.