LATS2 knockdown promoted glioma cell viability, Erastin treatment suppressed cell viability and the variation tendencies of cell viability by sh‐LATS2/sh‐WWP2/oe‐CMTM5 transfection in erastin‐induced glioma cells mimicked those in glioma cells without erastin treatment (Figure 5F). The gene discussed is WWP2; the disease is central nervous system cancer.