demonstrated that ALO induces the production of autophagosome‐like vacuoles in cells.[38] Notably, both in vitro and in vivo analyses revealed that ALO treatment interferes with the activation of the Akt/mTOR pathway in NSCLC cells, while levels of Beclin‐1 and ATG7 increase, suggesting that ALO treatment induces autophagosome generation. The gene discussed is BECN1; the disease is non-small cell lung carcinoma.