Furthermore, the combination of ALO with ATG7 silencing did not further increase ROS levels or apoptosis compared with ATG7 silencing alone in H1299 and LLC cells (Figure S9A–D, Supporting Information), indicating that ALO‐induced ROS accumulation and apoptosis in NSCLC cells are mediated by autophagy inhibition. This evidence concerns the gene ATG7 and non-small cell lung carcinoma.