Then, multiple research groups validated Omomyc in several cancer models, where it halted tumor progression and even induced tumor eradication, independently of the tissue of origin or driving oncogenic lesions.214,407,412–422 These studies also underscored that MYC inhibition, contrary to prior belief, was well-tolerated by normal proliferative tissues that simply slowed down their proliferation rate.421. Here, MYC is linked to neoplasm.