Studies have shown that EBV and its encoded LMP1 can potentially alter glucose metabolism in NPC, through increasing the expression of vascular endothelial growth factor to stimulate angiogenesis [16], inhibiting glucose deprivation and metabolism to restore sensitivity to apoptosis induction [17], and decreasing monocyte migration and T cell activation to promote immune escape [18]. This evidence concerns the gene PDLIM7 and nasopharyngeal carcinoma.