Given our data, previously published studies on PCa cells indicating EMT transition as a function of docetaxel resistance [17, 35, 36] and the link between EMT and increased expression of CD95 and SSEA-4 [37, 38], we computed the EMT score reflecting the extent to which cells display epithelial or mesenchymal phenotype on a scale from −1 (Epi) to 1 (Mes), in all pre- and post-docetaxel-based therapy samples using a two-sample Kolmogorov–Smirnov test [39]. The gene discussed is FAS; the disease is posterior cortical atrophy.