FASLG and posterior cortical atrophy: In addition, given the increased sensitivity of docetaxel-resistant PC3 cells to CD95L we assume that treatment with this ligand may rewire the non-canonical, pro-survival CD95 signaling in docetaxel-resistant cells and induce canonical, apoptosis-triggering CD95L-CD95 signaling, representing potential therapeutic avenue for the therapy of advanced PCa.