Myo1c is an unconventional class I myosin and actin‐based motor protein that is vital for cellular functions, such as intracellular trafficking, cell adhesion, motility, and maintenance of membrane tension.[50, 51, 52, 53] Previous researches suggested a potential target of Myo1c for the treatment of liver fibrosis and podocytopathies.[54, 55] Myo1c was recently reported to participate in hepatic cell proliferation and the wound‐healing response.[54] Here, we identified Myo1c as a mediator of liraglutide‐induced keratinocyte proliferation, migration, and adhesion. The gene discussed is IMMT; the disease is Hepatic fibrosis.