Particularly, pathogenic variants in superoxide dismutase 1 (SOD1), TAR DNA‐binding protein (TARDBP), fused in sarcoma (FUS) and chromosome 9 open reading frame 72 (C9orf72) genes, account for approximately 60% of the familial cases and about 10% of sporadic ALS (Akçimen et al., 2023). Here, SOD1 is linked to amyotrophic lateral sclerosis.