Figure 7c suggests that SEC + LipoMin also maintained a significantsignal for EV (Flotillin) and AD markers (phosphorylated tau at threonine217; p-tau217) and neuronal protein (NCAM).51,52Figure 7d shows UCand UC + LipoMin where UC + LipoMin maintained solid WB signals forp-tau217, NCAM, and Flotillin. This evidence concerns the gene NCAM1 and Alzheimer disease.