Increases apoptotic relevant protein BAX and caspase-9 expressions in H. pylori infection.Affects the levels of BCL-2 and BAX to protect against apoptosis associated with H. pylori infection.Improves mitochondrial function by restoring the level of ATP and MMP, inhibits H. pylori induced apoptosis.Suppresses the activation of apoptosis associated proteins induced by H. pylori.Inhibits intrinsic apoptosis proteins including BAX, cytochrome c, and caspase-3 mediated by infection. This evidence concerns the gene BCL2 and infection.