TET3 and endometriosis: While it is yet to be determined which origins of macrophages (eutopic endometrial tissue, infiltrating LPMs, and/or monocytes) undergo TET3 overexpression during endometriosis in mice, the myeloid-specific Tet3-KO is anticipated to eliminate the majority of TET3 OE macrophages irrespective of their developmental origins, other molecular features, and surface/intracellular markers, with the exception of those from donor mouse endometrium.