Helminth infections typically induce modified type 2 immune responses, characterized by the release of effector cytokines interleukin (IL-) 4, IL-5, and IL-13 from Th2 cells and type 2 innate lymphoid cells (ILC), and by heightened production of anti-inflammatory IL-10 by regulatory T (Treg) cells. Here, IL13 is linked to helminthiasis.