The results confirmed that LCN2 expression mainly focused on the alveolar epithelium and inflammatory cells of lung injury mice (Figures 1(d) and 1(e)), which was consistent with a previous study on LCN2 expression in chronic lung diseases, demonstrating that LCN2 was significantly upregulated in lung tissue after exposure to damage factors, such as ultrafine carbon particles, pathogens, and inflammatory cytokines [7, 8, 18]. The gene discussed is LCN2; the disease is chronic lung disease.