MAP1LC3B and small cell lung carcinoma: The results showed that the autophagy level was improved in H69AR cells compared with H69 cells and that the high level of autophagy may be a positive regulator of cell viability and may be involved in SCLC cell chemoresistance, as we previously reported.[20] PD (0.5 μm) or LEE (0.8 μm) treatment induced double‐positive LC3B puncta in SCLC cells, which indicated that PD and LEE treatment induced impaired autophagic flux in SCLC cells (Figure 4D; Figure S4D, Supporting Information).