As the major contributor of potential inflammatory storm (Figure 3), many inflammatory response genes (e.g., ITGB2, OSM, FPR1, CEBPB, NINJ1) and canonical pro‐inflammatory cytokines (e.g., CXCL8, CCL3, CCL4, TNF, IL1B, S100A12) were expressed at higher levels in brucellosis patients than in healthy donors (Figure 7F, Figure S10C). Here, IL1B is linked to brucellosis.