ATG9A and asthma: demonstrated in the mouse model of asthma induced by house dust mites that knocking out ULK1 gene can reduce the infiltration of inflammatory cells in lung tissue, restore the imbalance of Th1/Th2 ratio, and inhibit the formation of inflammasome, and the researcher suggested that inhibiting the activation of the ULK1/Atg9a/Rab9 signaling pathways can inhibit Golgi apparatus fragmentation and mitochondrial oxidative stress in asthma while reducing the production of NLRP3-mediated pulmonary epithelial inflammation (118).