This finding is in line with a previous study, which showed that combined inhibition of MEK1/2 plus autophagy had a synergistic anti‐proliferative effect in pancreatic ductal adenocarcinoma cell lines, which display aberrant K‐RAS activity, as well as patient‐derived pancreatic ductal adenocarcinoma xenograft tumors in mice [100]. The gene discussed is KRAS; the disease is pancreatic ductal adenocarcinoma.