On the contrary, glucose intolerance in Alx3-deficient mice reversed when animals were fed with HFD (Fig. 2F), showing blood glucose levels similar to those observed in SCD-fed wild type mice (compare Fig. 2F with 2E) and significantly lower than those observed in wild type mice fed on a HFD (Fig. 2G). This evidence concerns the gene ALX3 and Schnyder corneal dystrophy.