GOT2 and metabolic dysfunction-associated steatotic liver disease: One of the critical events in the progression of NAFLD is lipotoxicity, which arises from an excessive influx of FFAs into hepatocytes.25,26 Fatty acid-binding proteins transport FFAs to cell compartments and play a crucial role in cellular functions.26,27 These data illuminate the role of FABPs in hepatosteatosis and provide a rational basis for our findings.