A different molecular mechanism of SAN-induced inhibition of GC cell proliferation was studied by Dong et al. According to their study, SAN’s antitumor function in BGC-823 GC cells was influenced by the expression of miRNAs miR-96-5p and miR-29c-3p (miRNAs) and the mitogen-activated protein kinase (MAPK) signaling pathway, whose activation is associated with cell cycle arrest and apoptosis induction. This evidence concerns the gene WNK2 and gastric cancer.