A potential net decrease in WNT signaling due to FF with excessive protein intake and the absence of FTO- and ZFP217-targeting milk miRNAs may finally enhance the total number of adipocyte precursor cells, a critical disturbance during postnatal adipocyte development, priming early steps of aberrant adipogenesis, which may increase the risk of obesity later in life (Figure 2). The gene discussed is FTO; the disease is obesity due to melanocortin 4 receptor deficiency.