HT is well characterized by the production of autoantibodies like the most common antithyroid peroxidase antibodies and anti-thyroglobulin antibodies and with lymphocyte infiltration.[3] The symptoms of HT patients are various but not specific, including fatigue, more sensitivity to coldness, weakness and increase in body weight, which may be due to extensive involvement of more systems.[4] Patients with HT show distinct manifestations and pathophysiologic features depending on the severity of HT. This evidence concerns the gene TG and hematocrit.