CDKN1A also interacts with proteins involved in cell apoptosis, such as the Bcl‐2 family inhibitor, Bax, which promotes tumor cell apoptosis.[7] Furthermore, studies have shown that p53 transcriptionally inhibits SLC7A11, a key cysteine/glutamate reverse transporter protein, leading to inhibition of cysteine uptake and increased sensitivity of cells to ferroptosis.[8] Although p53 is highly mutated in different tumor types, its functions are often repressed in many tumors with wild‐type (WT) p53, making tumor cells resistant to chemotherapy. Here, BCL2 is linked to neoplasm.