For example, Chen et al. [154] proposed that various mesenchymal stem cells of different origins, such as normal mucosa-derived MSCs (N-OMSC), dysplasia-derived MSCS (D-OMSC), cancer-derived MSCs (C-OMSC), and the corresponding BM-MSCs, may be involved in tumour formation in oral carcinogenesis by inhibiting T CD3+ and CD45+ cell numbers and proliferation. This evidence concerns the gene PTPRC and neoplasm.