The findings that antineoplastic agents promote the mobilization of CD61+, Gr-1+CD11b+, and VEGFR2+ BMDCs in the circulating blood and increase the recruitment and retention of GFP+ BMDCs in tumor tissues of bone-marrow-transplanted mice could therefore at least partly explain the phenomenon of tumor angiogenesis enhancement by LDM chemotherapy. The gene discussed is ITGB3; the disease is neoplasm.