IL17A and metabolic dysfunction-associated steatotic liver disease: Pro-inflammatory diets may genetically interfere with hepatic β-oxidation, resulting in a massive upregulation of pro-inflammatory molecules (e.g., IL-1β, IL-17, and IL-18) and oxygen-responsive substances, which in turn increases the production of endogenous lipids and increases the risk of NAFLD, as well as exacerbating the damage related to chronic disease (1, 29, 30).