Notably, he did not have diabetes, tested negative for human immunodeficiency virus (HIV), and lacked chronic illnesses compromising his immune system. Diabetes can increase the risk of FG because it can lead to immunosuppression, poor wound healing, and defective phagocytosis. For diabetic patients, the use of SGLT2 inhibitors can lead to the development of FG, though the exact mechanism is unknown. The gene discussed is SLC5A2; the disease is diabetes mellitus.