The mechanism may be related to the upregulated expression of xeroderma pigmentosum group A (XPA), xeroderma pigmentosum group C (XPC), excision repair cross-complementation group 1 (ERCC1), replication protein A1 (RPA1), and replication protein A2 (RPA2) in the nucleotide excision repair (NER) pathway, which promotes DNA damage repair [42]. This evidence concerns the gene RPA2 and xeroderma pigmentosum.