In a study on glioblastoma, investigators used a biotinylation approach to search for the TAZ-binding proteins in glioblastoma cells and identified the NONO protein, which was essential for TAZ to form phase-separated condensates in the nucleus and to enhance the interaction between TAZ, TEAD, and Pol II subunit B1, ultimately leading to the enhanced transcription of oncogenic genes[89]. This evidence concerns the gene NONO and glioblastoma.