ALB and liver dysplastic nodule: Initial stages of DN manifest with glomerular hyperfiltration and hypertrophy, later progressing to impaired glomerular filtration barrier, heightened urinary albumin excretion, and accumulation of excessive extracellular matrix (ECM), leading to thylakoid stroma accumulation, hypertrophy, and consequent nodular glomerulosclerosis and interstitial fibrosis, culminating in ESRD and renal failure (4, 5).